Day 031 - 05 Oct 94 - Page 28


     
     1        them?
     2        A.  Sure, that is correct.
     3
     4   MS. STEEL:   So, your view is that although you consider that
     5        the data on individuals is weak, that does not detract
     6        from the overall parallels that can be drawn?
     7        A.  I do not think it does, because I think I have already
     8        explained the reasons why the individual data has inherent
     9        weaknesses in it because of the polygenetic makeup.  It is
    10        very interesting.  If I can give you an example of this,
    11        there is an enzyme system which is genetically determined
    12         -- I shall not give you the full name of because it is a
    13        long winded name, but it is abbreviated as AHH.  What this
    14        enzyme does is to break down polycyclic hydrocarbons.  If
    15        you are genetically -- it is under polygenetic control;
    16        that is, a number of genes are required for this
    17        particular enzyme system.  This is quite well established
    18        in that people who have a high activity of this enzyme,
    19        for that reason, are at particular risk and exaggerated
    20        risk to lung cancer.  I think this is an example of the
    21        way in which the wide diversity of genetic performance can
    22        influence risk to disease.
    23
    24   Q.   Just before we leave this page, the Berzelius Symposium --
    25        you have not yet managed to find the paper on that --
    26        perhaps you could explain a little bit about what was
    27        under debate, what exactly was debated at this symposium?
    28        A.  It was a debate really held by the Swedish Medical
    29        Society in Stockholm.  It had an international
    30        attendance.  Basically, it asked the question, if we are
    31        going to try to prevent heart disease, are we going to
    32        have a different diet to the diet that we are going to
    33        recommend for the prevention of cancer?  By and large,
    34        although there were some dissenters to the view, which is
    35        usually the case, by and large, I think everybody agreed
    36        that the recommendations that are prudent for the
    37        prevention of heart disease and cancer would be similar.
    38        But that was some time ago, and I think we have moved on
    39        since then.
    40
    41        I would suggest to you that the sort of recommendations
    42        that came out of the Scottish Office are something that
    43        one pays more attention to than what we did in Stockholm
    44        in 1986.  I think there is a considerable strength in the
    45        sort of report such as came out of the Scottish Office
    46        because they are unbiased; whereas, with the best will in
    47        the world, individuals, I dare say including myself, will
    48        have some form of personal bias both in terms of the
    49        evidence they have individually seen through their life,
    50        and maybe their own feelings about things that really have 
    51        nothing do with the evidence.  But when you get a group of 
    52        people together discussing the matter and trying to 
    53        formulate words that are agreed across a body of expert
    54        opinion, then I think organisations like the British
    55        Nutrition Foundation and the Scottish Office represent
    56        fairly unbiased but, nonetheless, critical views, critical
    57        views which probably hold back a bit from what people like
    58        myself might be wanting to recommend.
    59
    60   Q.   They were being cautious?

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